Ipratropium Bromide

CAS No. 22254-24-6

Ipratropium Bromide ( Ipratropium Bromide )

Catalog No. M13553 CAS No. 22254-24-6

Ipratropium Bromide is an antagonist of M3 type muscarinic acetylcholine receptors, used for the treatment of chronic obstructive pulmonary disease (COPD).

Purity : >98%(HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
50MG 34 In Stock
100MG 43 In Stock
200MG 74 In Stock
500MG Get Quote In Stock
1G Get Quote In Stock

Biological Information

  • Product Name
    Ipratropium Bromide
  • Note
    Research use only, not for human use.
  • Brief Description
    Ipratropium Bromide is an antagonist of M3 type muscarinic acetylcholine receptors, used for the treatment of chronic obstructive pulmonary disease (COPD).
  • Description
    Ipratropium Bromide is an antagonist of M3 type muscarinic acetylcholine receptors, used for the treatment of chronic obstructive pulmonary disease (COPD).
  • Synonyms
    Ipratropium Bromide
  • Pathway
    Endocrinology/Hormones
  • Target
    AChR
  • Recptor
    M3 mAChR
  • Research Area
    Neurological Disease
  • Indication
    ——

Chemical Information

  • CAS Number
    22254-24-6
  • Formula Weight
    412.37
  • Molecular Formula
    C20H30BrNO3
  • Purity
    >98%(HPLC)
  • Solubility
    DMSO:83 mg/mL (201.27 mM); Ethanol:83 mg/mL (201.27 mM); Water:83 mg/mL (201.27 mM)
  • SMILES
    CC([N+]1(C)C2CC(OC(C(C3=CC=CC=C3)CO)=O)CC1CC2)C.[Br-]
  • Chemical Name
    (endo,syn)-(±)-3-(3-Hydroxy-1-oxo-2-phenylpropoxy)-8-methyl-8-(1-methylethyl)-8-azoniabicyclo[3.2.1]octane bromide

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. Zhang W, et al. Eur J Pharmacol,010, 647(1-3), 178-187.
molnova catalog
related products
  • Cyclopentolate Hydro...

    Cyclopentolate Hydrochloride is a muscarinic acetylcholine receptors antagonist with Kis of 1.62 27.5 and 2.63 nM for M1 M2 and M3 receptors respectively.

  • CCMI

    CCMI is a potent and selective modulator of α7 nAChR-positive allosteric.

  • Thymidine

    Thymidine overload due to Thymidine phosphorylase deficiency, and mitochondrial toxicity caused by antiviral Thymidine analogues.